Corrected QT-interval and QT-dispersion indicate ventricular repolarization time and heterogeneity. Increased corrected QT and/or QT-dispersion are known to be the cause of ventricular arrhythmia in various systemic diseases and lead to increase in mortality and morbidity [5-7]. Knorr et al. demonstrated ciprofloxacin-induced QT-interval prolongation in patients diagnosed with IBD [8]. The reports regarding QT-interval and QT-dispersion in IBD with patients are inadequate.

Corrected QT-interval and QT-dispersion indicate ventricular repolarization time and heterogeneity. Increased corrected QT and/or QT-dispersion are known to be the cause of ventricular arrhythmia in various systemic diseases and lead to increase in mortality and morbidity [5-7]. Knorr et al. demonstrated ciprofloxacin-induced QT-interval prolongation in patients diagnosed with IBD [8]. The reports regarding QT-interval and QT-dispersion in IBD with patients are inadequate.. women with PCOS for improved resources. Frontline therapy being scarcely full proof, hence there is a resurgent trend towards immunotherapy. The efficacy of the antibodies that targets specific molecules expressed by tumor cells can be increased by attaching toxins to them [53] . Existing immunotoxins are based on bacterial toxins like pseudomonas exotoxin, plant exotoxin like ricin or radio-nucleotides. The toxins are chemically conjugated to a specific ligand such as the variable domain of the heavy or light chain of the monoclonal antibody. Normal cells lacking the cancer specific antigens are not targeted by the targeted antibody [54] . The most promising immunotoxin is the EGF fusion protein DAB389EGF, which is a fused EGF specific sequence and diphtheria toxin and have been found to be effective in EGFR over expressing breast tumor and non small cell lung cancer. The use of therapeutic antibody for the development of antibody-drug conjugate has been tried to improve the therapeutic potential e.g. Tositumomab by GlaxoSmithcline [55] , Anti-Tac(Fv)-PE38(LMB-2) against CD25, in B, T cell lymphoma and anti-B4-bR against CD19 in B-Non Hodgkins lymphoma [56] . As molecular studies of cancer have started revealing an increased epitope repertoire due to great strides in genomics and proteomics, the search for more effective Antibody-drug conjugate has got a spur in recent times [57] .

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